| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8479110 | Neurochemistry International | 2015 | 5 Pages |
Abstract
Our results demonstrate that CRF and UCN I inhibit the glutamate release in the hippocampus via CRFR1 and that CRFR2 does not participate to this process. Based on the previous and the present results we conclude that CRFR1 agonists can activate the HPA axis not only directly, but also indirectly by increasing the amygdalar GABA release and decreasing the hippocampal glutamate release.
Related Topics
Life Sciences
Biochemistry, Genetics and Molecular Biology
Cell Biology
Authors
Zsolt Bagosi, Beáta Balangó, Dávid Pintér, Krisztina Csabafi, Miklós Jászberényi, Gyula Szabó, Gyula Telegdy,