Article ID Journal Published Year Pages File Type
8524976 Biomedicine & Pharmacotherapy 2018 8 Pages PDF
Abstract
Our data indicated that the high level of HDAC6 inhibited miR-148b via maintaining the low acetylation of histones H3 and H4 in the miR-148b promoter, thus rescuing Ezrin expression and promoting CHOP resistance in DLBCL.
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Related Topics
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