| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8534901 | Life Sciences | 2018 | 40 Pages |
Abstract
The BDP/FOR inhibition degree was related to the inflammatory mediator- and cell-type considered. FOR additively enhanced BDP effects by partially involving both dependent- and independent-PKA mechanisms. Our results might contribute to highlight the strong relationship between specific molecular pathways and different sensitivity to the corticosteroid/β2-agonist effects and to clarify the molecular mechanisms underlying the BDP/FOR anti-inflammatory activity in vivo.
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Authors
Silvana Cianchetti, Cristina Cardini, Alessandro Corti, Marta Menegazzi, Elena Darra, Eleonora Ingrassia, Alfonso Pompella, Pierluigi Paggiaro,