| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8545689 | Environmental Toxicology and Pharmacology | 2018 | 14 Pages |
Abstract
In conclusion, exposure to DEE from B7, which was associated with higher exposure to polycyclic aromatic hydrocarbons, resulted in decreased number of T and NK lymphocytes, while DEE from SHB20 induced a higher level of DNA single-strand breaks, oxidative stress and increased red blood cells parameters. Additionally, DPF technology generated increased number of smaller PM and made the DEE more reactive and more harmful, manifested as deregulation of redox balance.
Keywords
MCHDEPGSSGRBCALTWBCEPOGPXGSHHIF-1PMNDPFFPGMCHCMIDHVOCREPBMCPAHLyMDEESSBTASROSASTAspartate aminotransferaseAlanine aminotransferaseAir pollutionALPAlkaline phosphataseerythropoietinShbChoCarbonyl compoundstriglyceridered blood cell countOxidative stressDiesel exhaust particlesHydrotreated vegetable oilSODperipheral blood mononuclear cellsNatural killer cellsSuperoxide dismutaseWhite blood cell counthypoxia-inducible factor 1Red cell distribution widthHemoglobin concentrationDiesel Particle FilterLymphocytespolymorphonuclear leukocytesMean corpuscular haemoglobinFAME یا fatty acid methyl esters fatty acid methyl estersMean corpuscular haemoglobin concentrationHgbHydrocarbonsPolycyclic aromatic hydrocarbonsTotal antioxidant statusreduced glutathionecreatininecholesteroloxidized glutathioneglutathione reductaseglutathione peroxidaseReactive oxygen species
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Authors
K. Dziendzikowska, M. Gajewska, J. Wilczak, R. Mruk, M. Oczkowski, E. Å»yÅa, T. Królikowski, M. StachoÅ, J. Ãvrevik, O. Myhre, M. Kruszewski, M. Wojewódzka, A. Lankoff, J. Gromadzka-Ostrowska,