| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8626356 | Placenta | 2018 | 6 Pages |
Abstract
LA blocks TNF and thrombin induced FM weakening at two points, inhibiting both GM-CSF production and downstream action. Thus, we speculate that LA may be a potential standalone therapeutic agent, or supplement to current therapy for prevention of pPROM related spontaneous preterm birth, if preclinical studies to examine feasibility and safety during pregnancy are successfully accomplished.
Related Topics
Life Sciences
Biochemistry, Genetics and Molecular Biology
Developmental Biology
Authors
Deepak Kumar, Robert M. Moore, Anudeepa Sharma, Brian M. Mercer, Joseph M. Mansour, John J. Moore,