| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8628661 | Cytokine | 2018 | 7 Pages |
Abstract
CS sampling of NT-proBNP, CA 125 and pro-anti-inflammatory cytokines provides an additional insight into the possible mechanisms by which these biomarkers lead to left ventricular remodeling. Our results clearly suggest that serum NT-proBNP and CA 125 levels not only in PV, but also in CS of patients with CHF, may be dependent on inflammation as a consequence of cytokine network activation.
Keywords
LVESVNT-proBNPCA 125LVEFperipheral venousChFLVEDVIQRCrtcarbohydrate antigen 125inflammationstandard deviationinterleukintumor necrosis factor-alphaLeft ventricular end-diastolic volumeCardiac resynchronization therapyCytokinescoronary sinusTNF-αinterquartile rangeheart failureChronic heart failureleft ventricular ejection fractionNYHA classNew York Heart Association class
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Authors
Adina Elena Stanciu, Marcel Marian Stanciu, Radu Gabriel Vatasescu,