Article ID Journal Published Year Pages File Type
8629735 Diabetes Research and Clinical Practice 2018 24 Pages PDF
Abstract
Our study suggests that miR-487a-3p might repress CTLA4 and FOXO3 by binding to their 3'UTRs and contribute to the development of T1D.
Related Topics
Life Sciences Biochemistry, Genetics and Molecular Biology Endocrinology
Authors
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