| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8632985 | Metabolism | 2018 | 25 Pages |
Abstract
These findings strengthen the involvement of AGEs-RAGE axis in liver injury and the pathogenesis of NAFLD.
Keywords
FFAhsCRPIL-8RAGEAGEsCMLHOMA-IRALTLC-MSγGTHPLC-MSNBTNFPAROCsRAGEPDACelDMFesRAGETGF-β1NAFLDHomeostasis model of assessment-insulin resistanceM30PBSIL-6AUCROSnitroblue tetrazoliumphotodiode arrayASTAspartate aminotransferaseAlanine aminotransferaseNon-alcoholic steatohepatitisFree fatty acidinterleukin 6Interleukin 8cardiovascular diseasenon-alcoholic fatty liver diseaseBiomarkertransforming growth factor beta 1Enzyme-linked immunosorbent assayELISACVDLSMbody mass indexBMICoefficient of Variationtumor necrosis factor alfaTNF-αphosphate buffer salineAdvanced glycation end productsInsulin resistancearea under the curveodds ratioNash Liverhigh-performance liquid chromatographyglutamyl transpeptidaseGlycationReactive oxygen speciesreceiver operating characteristic
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Authors
Susana A. Palma-Duran, Meropi D. Kontogianni, Antonis Vlassopoulos, Shudong Zhao, Aikaterini Margariti, Michael Georgoulis, George Papatheodoridis, Emilie Combet,