| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8633193 | Neuropeptides | 2018 | 10 Pages |
Abstract
Distinct physiological patterns of E secretion characteristic of the female rat estrous cycle elicit differential corticosterone outflow during hypoglycemia, and establish both common and different hypothalamic metabolic neurotransmitter targets of hindbrain lactate deficit signaling. Outcomes emphasize a need for insight on systems-level organization, interaction, and involvement of E signal strength-sensitive neuropeptides in counter-regulatory functions.
Keywords
pro-opiomelanocortinRPOaCSFAVPVnNOSAMPKARHMCHLHAOVXCV4PVHIIHVMHNPHGAD65/67Neutral protamine Hagedorn insulinGnRHTBSAICARSDSINSCRHPOMCMPNDMHNPYL-lactatepAMPKEstradiolinsulinorexin AOvariectomyTris-buffered salinesubcutaneoussodium dodecyl sulfateneuronal nitric oxide synthaseartificial cerebrospinal fluidlateral hypothalamic areanorepinephrinenitric oxide synthasedorsomedial hypothalamic nucleusVentromedial hypothalamic nucleusarcuate hypothalamic nucleusparaventricular hypothalamic nucleusanteroventral periventricular nucleusmedial preoptic nucleuscorticotropin-releasing hormonegonadotropin releasing hormoneluteinizing hormonemelanin-concentrating hormoneInsulin-induced hypoglycemiarecurrent insulin-induced hypoglycemia5′ adenosine monophosphate-activated protein kinaseNeuropeptide Y
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Authors
Santosh K. Mandal, Prem K. Shrestha, Fahaad S.H. Alenazi, Manita Shakya, Hussain N. Alhamami, Karen P. Briski,