| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8645183 | Gene | 2018 | 29 Pages |
Abstract
The interplay between NOD2 signaling, proinflammatory cytokines, CHIT1 activity, heightened oxidative stress and DNA damage orchestrates allergic airway inflammation and thus contributing to the pathogenesis of atopic asthma. These parameters qualified for measurement as part of new noninvasive biomarker panels for monitoring asthma severity.
Keywords
FEV1MDAGSTPAMPsNucleotide-binding oligomerization domain 2CHIT1FVCNOD2NLR3-NT3-nitrotyrosineROSDNA damagepathogen-associated molecular patternsforced expiratory volume in 1 sproinflammatory cytokinesforced vital capacitymalondialdehydeglutathione-S-transferaseReactive oxygen speciesNod-like receptor
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Authors
Hanaa H. Gaballah, Rasha A. Gaber, Ragia S. Sharshar, Samah A. Elshweikh,