| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8647658 | Multiple Sclerosis and Related Disorders | 2017 | 7 Pages |
Abstract
- An impaired cell-mediated immunity is common in tumefactive demyelinating lesions.
- An impaired cell-mediated response to pathogens could lead to their persistence.
- Persistence of noxa pathogena stimulates a compensatory innate immune response.
- Immunothrombosis is a procoagulant innate immune mechanism against pathogens.
- Immunothrombosis might be at the basis of the tumefactive demyelinating lesions.
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Authors
Tatiana Koudriavtseva, Svetlana Lorenzano,