Article ID Journal Published Year Pages File Type
8648439 Molecular Immunology 2018 6 Pages PDF
Abstract
The present results suggest that hypoxia is capable of promoting the Th17 response by reducing AhR activity via HIF-1α activity. Thus hypoxia may be intimately involved in the pathogenesis of AR.
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Life Sciences Biochemistry, Genetics and Molecular Biology Molecular Biology
Preview
Hypoxia disrupts aryl hydrocarbon receptor signaling and the Th17 response in allergic rhinitis patients
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