| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8651852 | The American Journal of the Medical Sciences | 2017 | 30 Pages |
Abstract
TUC40- overexpression reduced Pbx1 expression, cardiomyocyte induction and differentiation, inhibited proliferation and promoted apoptosis. Combining the results and previous studies, we propose TUC40- as a potential pathologic factor for VSD.
Related Topics
Health Sciences
Medicine and Dentistry
Cardiology and Cardiovascular Medicine
Authors
Huijuan Li, Li PhD, Zhangbin PhD, Shuping PhD, Xuehua Liu, Mengmeng Li, Chun Zhu, Lixing PhD, Li PhD,