| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8661688 | International Journal of Cardiology | 2018 | 7 Pages |
Abstract
The identification of novel key players may expand our perspectives and promote the development of new tools for future preventive and therapeutic strategies in order to reduce the adverse vascular remodeling following PCI. The latter represents one of the major goals in the development of innovative technologies with relevance for clinical practice.
Keywords
BMSOXPHOSICAM-1cGMPNOSMCP-1eNOSISRGSK-3βNF-κBPGC-1αGADPHPDGFMFNPCIEESoxLDLEverolimus eluting stentflavin adenine dinucleotideH2SNADPHmRNAPESBESMitochondrial DNAopa1MAPKO2−Hydrogen peroxideoptic atrophy 1Adenosine TriphosphateATPsuperoxide anionDESDrug-eluting stentsIn-stent restenosisBare-metal stentendothelial NOSFADmtDNAmessenger ribonucleic acidHydrogen sulfideplatelet derived growth factornuclear factor kappa BOxidative phosphorylationOxidized low-density lipoproteinsMEKPercutaneous coronary interventionsintercellular adhesion molecule-1cyclic guanosine monophosphateMitofusinNitric oxidenitric oxide synthasenicotinamide adenine dinucleotide phosphateH2O2monocyte chemoattractant protein-1dynamin-related protein 1mitogen-activated protein kinasePaclitaxel-eluting stentGlycogen synthase kinase-3β
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Authors
Giovanna Gallo, Giorgia Pierelli, Maurizio Forte, Roberta Coluccia, Massimo Volpe, Speranza Rubattu,