| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8661782 | International Journal of Cardiology | 2018 | 7 Pages |
Abstract
Our study suggests that the interaction between TLR2 and HMGB1 contributes to the pathogenesis of cardiac fibrosis via suppressing fibroblast autophagy, and that inhibiting HMGB1 with GA provides therapeutic benefits for the treatment of fibroproliferative heart diseases.
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Authors
Ri-Na Wu, Tian-Yu Yu, Ji-Chao Zhou, Meng Li, Hui-Kuan Gao, Can Zhao, Rui-Qing Dong, Dian Peng, Zhuo-Wei Hu, Xiao-Wei Zhang, Yong-Quan Wu,