| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8661901 | International Journal of Cardiology | 2018 | 7 Pages |
Abstract
Our study suggested that metabolic changes in the atrial myocardium caused mitochondrial oxidative stress and potential cell damage, which further led to atrial fibrosis and myofibril disorganization, the characteristic phenotype of IIF-ACM.
Keywords
NSRITRAQPCAscxTemHistologyisobaric tags for relative and absolute quantitationAtrial tachycardiastrong cation exchangePrincipal component analysisOxidative stressKEGG یا Kyoto Encyclopedia of Genes and Genomes right anterior obliqueRAONormal sinus rhythmFibrosisAtrial fibrillationMetabolismTransmission electron microscopyProteomics
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Authors
Chang Cui, Xiaohong Jiang, Weizhu Ju, Jiaxian Wang, Daowu Wang, Zheng Sun, Minglong Chen,