Article ID Journal Published Year Pages File Type
8662547 International Journal of Cardiology 2018 7 Pages PDF
Abstract
It is proposed that DiOHF confers protection against myocardial reperfusion injury by inhibiting CaMKII and subsequent PLN-induced leak of Ca2 + from the sarcoplasmic reticulum as well as by inhibiting JNK2 activation to reduce apoptosis.
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Related Topics
Health Sciences Medicine and Dentistry Cardiology and Cardiovascular Medicine
Preview
Protection against reperfusion injury by 3′,4′-dihydroxyflavonol in rat isolated hearts involves inhibition of phospholamban and JNK2
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