| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8674266 | Molecular Metabolism | 2018 | 8 Pages |
Abstract
Our data provide initial mechanistic insights underlying the Pi-dependent regulation of Fgf23 secretion in identifying PiT2 as a potential player in this process, at least in high Pi conditions. Targeting PiT2, therefore, could improve excess FGF23 in hyperphosphatemic conditions such as chronic kidney disease.
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Authors
Nina Bon, Giulia Frangi, Sophie Sourice, Jérôme Guicheux, Sarah Beck-Cormier, Laurent Beck,