| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8679258 | Thrombosis Research | 2018 | 9 Pages |
Abstract
MI induces the release of different EVs subpopulations, including those of cardiac origin, in a preclinical model of MI and STEMI patients. In vitro, cardiomyocyte derived EVs are able to modulate endothelial function, suggesting their active role in heart repair after ischemia.
Keywords
ECMEVSFACSMicrovesiclesMmpsVCAMICAMSTEMIPFPRT-PCRECsH&EMyocardial infarctionIntercellular communicationEndotheliumST-segment elevation myocardial infarctionIschemiaCardiac repairExtracellular vesiclesEndothelial cellsFlow cytometrycardiovascularLADExtracellular matrixMatrix metalloproteinasesintercellular adhesion moleculevascular cell adhesion moleculewild typehematoxylin eosinReal time polymerase chain reactionplatelet-free plasmaleft anterior descending
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Authors
Jose A. Rodriguez, Josune Orbe, Goren Saenz-Pipaon, Gloria Abizanda, Natalia Gebara, Flavia Radulescu, Pedro M. Azcarate, Luis Alonso-Perez, David Merino, Felipe Prosper, Jose A. Paramo, Carmen Roncal,