| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8681530 | Brain Stimulation | 2018 | 63 Pages |
Abstract
Advances in knowledge of the immuno-inflammatory component of depression seem to be paving the way towards models to explain the mechanism of action of ECT.
Keywords
SCL-90CRFpdpCMHPGTSHSADS-LNGFTIMPsCPRSS-100BRIAMCVPHA-PMCHCHAM-DAVPPRLBDIDSTMADRSMCHTSPOIDOQUINMmpsConcanavalin ATissue inhibitors of MMPsTNFTrpCRHECTNSELPSNKCAnuclear factor-kappa-BNF-kBUFCMDDRBCACTHcyclic AMPNeuron-specific enolaseCyclic adenosine monophosphateDepressive disorderMajor depressive disordersBipolar disorderskynurenic acidQuinolinic acidMajor depressioninflammationImmuneinterferonIFNinterleukineCon AtrhTryptophandexamethasone suppression testthyrotrophinDirect CurrentKYNMean corpuscular volumeelectroconvulsive treatmentCNSradioimmunoassaycentral nervous systemcorticotropin releasing factornerve growth factortumor necrosis factorNatural killer cell activitylipopolysaccharideCSFMatrix metalloproteinasesmean corpuscular hemoglobinMontgomery-asberg depression rating scaleHamilton Rating Scale for DepressionMean corpuscular hemoglobin concentrationMINInoradrenalineNitric oxideHemoglobinadrenocorticotropic hormonecorticotropin releasing hormonefollicle stimulating hormoneluteinizing hormoneGrowth hormoneFSHarginine vasopressinBeck Depression InventoryS-100B proteintranslocator proteinCRPProlactinhealthy controlCORTcortisolUrinary free cortisolKYNAkynureninered blood cell
Related Topics
Life Sciences
Neuroscience
Biological Psychiatry
Authors
Antoine Yrondi, Marie Sporer, Patrice Péran, Laurent Schmitt, Christophe Arbus, Anne Sauvaget,