IL-1/inhibitory ÎºB kinase Îµ-induced glycolysis augment epithelial effector function and promote allergic airways disease

Article ID	Journal	Published Year	Pages	File Type
8712914	Journal of Allergy and Clinical Immunology	2018	26 Pages	PDF

Keywords

AHR Nasal epithelial cell IL-1 receptor type I MTE LDHA HK2 Inhibitory κB kinase IL-1RI TSLP TBK1 HDM 2-Deoxyglucose NEC IKK ECAR BAL IL-1 mTOR 2-DG Small interfering RNA siRNA Asthma TANK-binding kinase 1 Dendritic cell body mass index BMI bronchoalveolar lavage Lactate Lactate dehydrogenase A Thymic stromal lymphopoietin Airway resistance extracellular acidification rate wild-type mammalian target of rapamycin hexokinase 2 airways hyperresponsiveness House dust mite Glycolysis

Related Topics

Life Sciences Immunology and Microbiology Immunology

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IL-1/inhibitory ÎºB kinase Îµ-induced glycolysis augment epithelial effector function and promote allergic airways disease

Authors

Xi PhD, Reem MS, Cheryl MS, Shi B. MS, Eyal PhD, Robert W. BS, Jos L.J. PhD, Karolyn G. BS, Daisy A. BS, Dylan T. BS, Nirav MS, Jennifer L. PhD, Matthew J. PhD, Minara BS, Kendall E. BS, David G. PhD, Lennart K.A. PhD, David H. PhD, Yvonne M.W. PhD,