| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8715743 | Journal of Dermatological Science | 2017 | 9 Pages |
Abstract
Our results indicate that ÎNp73 is an additional participant in the mechanism of TSLP production. Amending the aberrant state of keratinocytes, represented by overexpression of ÎNp73, can be a novel therapeutic target of AD.
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Authors
Ayako Kumagai, Terufumi Kubo, Koji Kawata, Ryuta Kamekura, Keiji Yamashita, Sumito Jitsukawa, Tomonori Nagaya, Yasuyuki Sumikawa, Tetsuo Himi, Toshiharu Yamashita, Shingo Ichimiya,