| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8726641 | Gastroenterology | 2018 | 15 Pages |
Abstract
We identified a mechanism by which the microbiota controls susceptibility to C jejuni infection in mice, via bacteria-derived secondary bile acids.
Keywords
DCAMLNUDCAmTORTCARDNAmRNASPFCFUPBSHigh-performance liquid chromatography/mass spectrometryPI3KRibosomal DNAmessenger RNALCADeoxycholic acidTaurocholic acidLithocholic acidCholic acidUrsodeoxycholic acidIHCImmunohistochemistryGerm freespecific pathogen-freeInfectionPhosphatidylinositol 3-kinasefluorescence in situ hybridizationFishMetabolismPhosphate-buffered salineMicrobiomemammalian target of rapamycincolony-forming unitpolymerase chain reactionPCRbody weightmesenteric lymph node
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Authors
Xiaolun Sun, Kathryn Winglee, Raad Z. Gharaibeh, Josee Gauthier, Zhen He, Prabhanshu Tripathi, Dorina Avram, Steven Bruner, Anthony Fodor, Christian Jobin,