Mitochondrial Dysfunction, Through Impaired Autophagy, Leads to Endoplasmic Reticulum Stress, Deregulated Lipid Metabolism, and Pancreatitis in Animal Models

Article ID	Journal	Published Year	Pages	File Type
8726910	Gastroenterology	2018	15 Pages	PDF

Keywords

CatB ADP CCK PTP i.p.LC-MS Cholecystokinin-8 FFA ΔΨm Cyclophilin D Free cytosolic Ca2+ concentration CypD GC-MS MPO [Ca2+]i Adenosine Triphosphate ATP adenosine diphosphate l-arginine Electrophoretic mobility shift assay Lamellar bodies Arg Free fatty acid Polyunsaturated fatty acid PUFA Permeability transition pore Immunoblot Immunofluorescence Immunohistochemistry IHC triacylglycerol TAG یا triacylglycerols standard error of the mean intraperitoneal Acinar cell EMSA یا electrophoretic mobility shift assay endoplasmic reticulum Liquid chromatography-mass spectrometry lipid droplet lamellar body SEM myeloperoxidase Electron microscopy knockout Inflammatory response Acute pancreatitis Pancreas Mitochondrial membrane potential perilipin Cathepsin B PLIN Gas Chromatography-Mass Spectrometry

Related Topics

Health Sciences Medicine and Dentistry Gastroenterology

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Mitochondrial Dysfunction, Through Impaired Autophagy, Leads to Endoplasmic Reticulum Stress, Deregulated Lipid Metabolism, and Pancreatitis in Animal Models

Authors

Gyorgy Biczo, Eszter T. Vegh, Natalia Shalbueva, Olga A. Mareninova, Jason Elperin, Ethan Lotshaw, Sophie Gretler, Aurelia Lugea, Sudarshan R. Malla, David Dawson, Piotr Ruchala, Julian Whitelegge, Samuel W. French, Li Wen, Sohail Z. Husain,