| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8727381 | Gastroenterology | 2017 | 15 Pages |
Abstract
Mice with defects in Paneth cells (induced by maternal separation or genetically engineered) have intestinal expansion of E coli leading to visceral hypersensitivity. These findings provide evidence that Paneth cell function and intestinal dysbiosis are involved in visceral sensitivity.
Keywords
CFUVIPIBSAMPPLS-DANECTNFαIFNγGvHDAUCStressElectromyographEMGNecrotizing enterocolitisvariable importance in projectioninterleukinGraft-versus-host diseaseCrohn’s diseasePartial least-squares discriminant analysisanalysis of varianceANOVAEnzyme-linked immunosorbent assayELISAtumor necrosis factor αmaternal separationAbdominal painirritable bowel syndromeAntimicrobial activitylysozymearea under the curvecolony-forming unitpolymerase chain reactionPCRAntimicrobial peptidesInterferon gamma
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Authors
Ambre Riba, Maïwenn Olier, Sonia Lacroix-Lamandé, Corinne Lencina, Valérie Bacquié, Cherryl Harkat, Marion Gillet, Marine Baron, Caroline Sommer, Virginie Mallet, Christel Salvador-Cartier, Fabrice Laurent, Vassilia Théodorou, Sandrine Ménard,