| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8730418 | Pancreatology | 2018 | 5 Pages |
Abstract
These data suggest that RGC-32 promotes the proliferation of pancreatic cancer and induces the epithelial-mesenchymal transition (EMT). It would be a future direction of research to investigate the regulatory mechanism of signal molecules downstream RGC-32 on EMT-related transcription factors and deliberate the role of RGC-32 in tumorigenicity. As a result, RGC-32 may become a new therapeutic target for cancers.
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Authors
Liang Zhu, Ying Ding,