Article ID Journal Published Year Pages File Type
8739563 Journal of Autoimmunity 2018 8 Pages PDF
Abstract
Our data support a contributory role of altered methylation mechanisms in the pathogenesis of systemic autoimmune disorders and related lymphoproliferative processes and suggest that LSH and DNMT3A should be investigated as candidate upstream mediators of decreased L1 promoter methylation and increased L1 expression.
Related Topics
Life Sciences Immunology and Microbiology Immunology
Authors
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