| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8780799 | Gynecologic Oncology | 2017 | 6 Pages |
Abstract
The TTC28-L1 transductions along with classical driver mutations were near ubiquitous across the tumors, suggesting that L1 activation likely occurred early in the development of EAOCs. TTC28-L1 transductions could potentially be used to determine clonal relationships and to track ovarian cancer progression.
Keywords
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Authors
Zhouchunyang Xia, Dawn R. Cochrane, Michael S. Anglesio, Yi Kan Wang, Tayyebeh Nazeran, Basile Tessier-Cloutier, Melissa K. McConechy, Janine Senz, Amy Lum, Ali Bashashati, Sohrab P. Shah, David G. Huntsman,