| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8807964 | Pathophysiology | 2017 | 9 Pages |
Abstract
Our findings suggest that a single i.v. dose of RGAE causes ACE dipeptidase inhibition, thus reducing AngII generation and bioavailability in the 2K-1Cr. This action of RGAE enhances the non-clipped kidney clearance of sodium and water by modulating the tubular handling mechanisms.
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Authors
Khaled K. Al-Qattan, Martha Thomson, Muslim Ali,