| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8814210 | Biological Psychiatry | 2018 | 42 Pages |
Abstract
The results are consistent with models implicating disrupted PFC-thalamic connectivity in the pathophysiology of schizophrenia and mechanisms of cognitive impairment. PFC-thalamic anatomical connectivity may be an important target for procognitive interventions. Further work is needed to determine the implications of increased thalamic connectivity with sensory cortex.
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Authors
Monica Giraldo-Chica, Baxter P. Rogers, Stephen M. Damon, Bennett A. Landman, Neil D. Woodward,