| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8814425 | Biological Psychiatry: Cognitive Neuroscience and Neuroimaging | 2018 | 50 Pages |
Abstract
Atypicality of N170-particularly latency-to faces appears to be a specific biomarker of social-communicative dysfunction in ASD and may relate to differential developmental experiences and use of compensatory cognitive mechanisms. Future research should examine phenotypic differences that contribute to N170 heterogeneity, as well as specificity of N170 differences in ASD versus non-ASD clinical populations, and N170 malleability with treatment.
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Authors
Erin Kang, Cara M. Keifer, Emily J. Levy, Jennifer H. Foss-Feig, James C. McPartland, Matthew D. Lerner,