Article ID Journal Published Year Pages File Type
8817633 Biomedical and Environmental Sciences 2017 8 Pages PDF
Abstract
The current study provided direct evidence that exogenous glutamate treatment impaired the mitochondrial respiration capacity of astrocytes and enhanced aerobic glycolysis, which could be involved in glutamate injury or protection mechanisms in response to neurological disorders.
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Health Sciences Medicine and Dentistry Public Health and Health Policy
Authors
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