Article ID Journal Published Year Pages File Type
8839861 Brain Research 2018 48 Pages PDF
Abstract
Our results suggest that BH4 and/or ALC might protect the brain against oxidative stress induced neuronal apoptosis by restoring bioavailability of BH4 and upregulating of BH4 synthetic enzymes in the brain during sepsis.
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Related Topics
Life Sciences Neuroscience Neuroscience (General)
Preview
Possible involvement of tetrahydrobiopterin in the disturbance of redox homeostasis in sepsis - Induced brain dysfunction
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