| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8931255 | British Journal of Anaesthesia | 2016 | 9 Pages |
Abstract
The development of organ dysfunction during human sepsis is linked to mitochondrial dysfunction, and so activation of PGC1α/NFE2L2 is likely to be beneficial. These pathways are attractive therapeutic targets for sepsis.
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Authors
G. McCreath, M.M.F. Scullion, D.A. Lowes, N.R. Webster, H.F. Galley,