Article ID Journal Published Year Pages File Type
8934282 British Journal of Anaesthesia 2013 7 Pages PDF
Abstract
Ketamine-induced neuroapoptosis is associated with a temporal decrease in GSK-3β phosphorylation, and simultaneous administration of lithium mitigated this response. These findings suggest that GSK-3β is activated during this ketamine-induced neuroapoptosis.
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Health Sciences Medicine and Dentistry Anesthesiology and Pain Medicine
Preview
Role of glycogen synthase kinase-3β in ketamine-induced developmental neuroapoptosis in rats
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