| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8935519 | British Journal of Anaesthesia | 2011 | 8 Pages |
Abstract
Both ischaemic and hypoxic hypoxia initiate glycocalyx degradation, promoting an increase in permeability. A contributing mechanism could be purine-mediated degranulation of resident mast cells, with liberated tryptase β acting as potential 'sheddase'.
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Authors
T. Annecke, J. Fischer, H. Hartmann, J. Tschoep, M. Rehm, P. Conzen, C.P. Sommerhoff, B.F. Becker,