| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8996305 | Medical Hypotheses | 2005 | 4 Pages |
Abstract
A proposed mechanism whereby the production of clavulanate may yield high levels of urea/ammonia in the child is presented. Further an examination of this mechanism needs to be undertaken to determine if a subset of children are at risk for neurotoxicity from the use of clavulanic acid in pharmaceutical preparations.
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Authors
Joan Fallon,