| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 8996489 | Medical Hypotheses | 2005 | 7 Pages |
Abstract
This paper examines specific epigenetic and biochemical mechanisms possibly mediated by VN or receptor genes resulting in postulated VN autoimmune fatigue-related disorders. These mechanisms may have implications for treatment and prevention options for VN autoimmune disorders. VN autoimmune processes have implications for military medicine where radiological, chemical and biological agents may play an important role in pathogenesis.
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Authors
Donald R Staines,