| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 9109441 | Placenta | 2005 | 8 Pages |
Abstract
We concluded that upregulation of sFlt-1 and unbalanced PlGF/VEGF production associated with increased oxidative stress are consequences of hypoxia in placental TCs. Our results suggest that placental TCs are major sources of sFlt-1 and VEGF levels in the maternal circulation in women with preeclampsia.
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Authors
H. Li, B. Gu, Y. Zhang, D.F. Lewis, Y. Wang,