Article ID Journal Published Year Pages File Type
9115691 Metabolism 2005 9 Pages PDF
Abstract
Our findings suggest that camostat prevents and reverses obesity, hyperinsulinemia, hyperglycemia, and hyperlipidemia and markedly inhibits inflammation, fibrosis, and disruption of the islets in the genetically obese diabetic OLETF rats.
Related Topics
Life Sciences Biochemistry, Genetics and Molecular Biology Endocrinology
Preview
Synthetic protease inhibitor camostat prevents and reverses dyslipidemia, insulin secretory defects, and histological abnormalities of the pancreas in genetically obese and diabetic rats
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