| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 9150042 | Physiology & Behavior | 2005 | 8 Pages |
Abstract
These data confirm previous reports indicating that CCK1 receptors mediate increased DVC Fos-LI following intestinal infusion of oleate or glucose. CCK1 receptors also contribute to increased Fos-LI in enteric neurons following intestinal oleate infusion. However, failure of lorglumide to attenuate the increase of Fos-LI in the ileal submucosal plexus following intestinal glucose suggests that some intestinal nutrients trigger Fos-LI induction via CCK1 receptor-independent pathways.
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Authors
Stephen Gulley, Mihai Covasa, Robert C. Ritter, Ayman I. Sayegh,