Fat Accumulation, Leptin, and Hypercapnia in Obstructive Sleep Apnea-Hypopnea Syndrome

Article ID	Journal	Published Year	Pages	File Type
9164117	Chest	2005	7 Pages	PDF

Abstract

These results suggest that the location of body fat deposits may not contribute to the pathogenesis of hypoventilation, and circulating leptin may fail to maintain alveolar ventilation in hypercapnic patients with OSAHS.

Keywords

OSAHS VFA SFA aHI Sao2 oxygen saturation Visceral fat accumulation Respiratory depression Obstructive sleep apnea-hypopnea syndrome apnea-hypopnea index body mass index BMI Vital capacity CSF Cerebrospinal fluid Obesity Visceral fat Subcutaneous fat

Related Topics

Health Sciences Medicine and Dentistry Cardiology and Cardiovascular Medicine

Preview

Fat Accumulation, Leptin, and Hypercapnia in Obstructive Sleep Apnea-Hypopnea Syndrome

Authors

Ryuhi MD, Koichiro MD, FCCP, Akira MD, Yasunori MD, FCCP, Nobuhiro MD, FCCP, Yuichi MD, FCCP, Takayuki MD, FCCP,