Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
9227433 | Journal of Allergy and Clinical Immunology | 2005 | 7 Pages |
Abstract
Initiation and regulation of allergic inflammation is influenced by many factors, including cell type, membrane receptors, and mediators generated. Furthermore, the altered response of targeted tissues (ie, airway smooth muscle) becomes important to the eventual expression of asthma. Finally, the genetic regulation and association of genetic polymorphisms has enhanced our understanding of host susceptibility. In this review key findings published in 2004 issues of the Journal of Allergy and Clinical Immunology are highlighted to demonstrate recent advances in these areas.
Keywords
eNOcytotoxic T-lymphocyte antigenEDNOX40LCTLAITIMTLRBALCysteinyl leukotrienesMIPAsthmaEosinophilsexhaled nitric oxideToll-like receptorAirway smooth muscleVascular endothelial growth factorVascular Endothelial Growth Factor (VEGF)bronchoalveolar lavageOX40 ligandMast cellsNitric oxidemacrophage inflammatory proteinprostaglandinSingle nucleotide polymorphismSNP
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Authors
Bruce S. MD, William W. MD,