| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 9308277 | Kidney International | 2005 | 13 Pages |
Abstract
A1 and A20 exert differential cytoprotective effects in RPTECs. A1 is antiapoptotic. A20 is anti-inflammatory via blockade of NF-κB. We propose that A1 and A20 are both required for optimal protection of RPTECs from apoptosis (A1) and inflammation (A20) in conditions leading to renal damage.
Related Topics
Health Sciences
Medicine and Dentistry
Nephrology
Authors
Uta Kunter, Soizic Daniel, Maria B. Arvelo, Jean Choi, Tala Shukri, Virendra I. Patel, Christopher R. Longo, Salvatore T. Scali, Gautam Shrikhande, Eduardo Rocha, E.V.A. Czismadia, Christina Mottley, Shane T. Grey, Jürgen Floege, Christiane Ferran,