| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 9308336 | Kidney International | 2005 | 7 Pages |
Abstract
The results point toward an important participation of tubular damage in the pathogenesis of this disease. It may also be suggested that in normotensive and normoalbuminuric ADPKD patients, a gradual increase of glomerular filtration would be produced. After the start of hypertension and microalbuminuria, the glomerular filtration rate (GFR) would decrease progressively, although more slowly.
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Authors
J. Antonio Casal, Jesús Hermida, Xosé M. Lens, J. Carlos Dr,