| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 9308552 | Kidney International | 2005 | 7 Pages |
Abstract
Our study demonstrates an independent role of the CCR5 Î32 genotype for the clinical outcome in IgA nephropathy. In vitro experiments revealed a reduced chemotactic response of monocytes from CCR5 Î32 carriers, thus pointing out a possible pathophysiologic explanation for the beneficial effect of the CCR5 Î32 genotype.
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Authors
U.L.F. Panzer, Andr Schneider, Oliver M. Steinmetz, Ulrich Wenzel, Petra Barth, Rdiger Reinking, J.A.N. U. Becker, Sigrid Harendza, Gunther Zahner, Michael Fischereder, Bernhard H. Kramer, Detlef Schlondorff, Tammo Ostendorf, Jrgen Floege,