Article ID | Journal | Published Year | Pages | File Type |
---|---|---|---|---|
9308997 | Kidney International | 2005 | 10 Pages |
Abstract
Inhibiting podocyte proliferation in experimental glomerulonephritis is associated with improvement in renal function and histology, suggesting that inhibiting CDK2 activity is a potential therapeutic target for glomerular diseases characterized by podocyte proliferation.
Related Topics
Health Sciences
Medicine and Dentistry
Nephrology
Authors
Siân V. Griffin, Ronald D. Krofft, Jeffrey W. Pippin, Stuart J. Shankland,