| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 9309099 | Kidney International | 2005 | 10 Pages |
Abstract
Overall, these findings suggest that CIC containing aberrantly glycosylated IgA1 affect proliferation of MC in vitro and, thus, likely play a role in the pathogenesis of IgAN.
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Authors
Jan Novak, Milan Tomana, Rhubell Brown, Stacy Hall, Lea Novak, Bruce A. Julian, Robert J. Wyatt, Jiri Mestecky, Karel Matousovic,