| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 9309119 | Kidney International | 2005 | 7 Pages |
Abstract
Our study demonstrates that iron overload is frequently present in renal transplant patients and shows a continuous decrease over time. This decrease is possibly impaired by the HFE C282Y and HFE H63D mutations. Furthermore, mutations in HFE may influence liver function as reflected by increased alanine-aminotransferase concentrations.
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Authors
Matthias Lorenz, Josef Kletzmayr, Andrea Huber, Walter H. Hörl, Gere Sunder-Plassmann, Manuela Födinger,