| Article ID | Journal | Published Year | Pages | File Type |
|---|---|---|---|---|
| 9310815 | Kidney International | 2005 | 6 Pages |
Abstract
These findings implicate a glomerulotubular cross-talk with mediators released from the mesangium, contributing to the pathogenesis of tubulointerstitial damage in IgAN. There are preliminary data to suggest that the expression of angiotensin II subtype-1 receptor and angiotensin II subtype-2 receptor in PTEC differs from that of HMC. These novel findings may provide clinicians new therapeutic approach for selective blockade of the tubulointerstitial injury in IgAN.
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Authors
Kar Neng Lai, Loretta Y.Y. Chan, Joseph C.K. Leung,